Yesterday I swung through a local party store to purchase a pint of cheap vodka.
Vodka makes a fine solvent for ivermectin and a "pint" is 375ml which is a convenient size to work with.
The woman who waited on me was clearly in the process of transitioning to a "man". She had a full beard.
The usual process seems to be to start with hormone therapy, which is relatively inexpensive, and then progress to the expensive surgeries where bits are moved around. I presume it is possible for a woman to spend years with one foot on the dock and the other in the canoe as doctors fight with insurance companies attempting to get the cost of the surgeries (or portions there-of) covered.
Depending on how quickly the process moves along, it is entirely possible for a bearded woman to conceive and have a child; even if she identifies as a "man".
Peer reviewed papers
Bonnie Auyeung et al published a paper in the British Journal of Psychology in 2009 where they reported the results of a longitudinal study where they sampled the amniotic fluid and then followed those children to adolescence. They sorted the group by testosterone level into high and low levels.
Let me point out that all of the mothers were both genetically and hormonally "women" and none were carpet-bombed with testosterone via shots or pills.
In this paper, we examine the possibility of a link between autistic traits and fT (fetal Testosterone) levels measured in amniotic fluid during routine amniocentesis. Two instruments measuring number of autistic traits (the Childhood Autism Spectrum Test (CAST) and the Child Autism Spectrum Quotient (AQ-Child)) were completed by these women about their children (N=235), ages 6–10 years...fT levels were positively associated with higher scores on the CAST and AQ-Child. This relationship was seen within sex as well as when the sexes were combined, suggesting this is an effect of fT rather than of sex per se.
Auyeung co-authored a follow-up paper in 2012 where brain activity imaging was performed on the children as they were exposed to various stimuli.
Increasing FT predicted enhanced selectivity for positive compared with negatively valenced facial cues in reward-related regions such as caudate, putamen, and nucleus accumbens but not the amygdala.
This work suggests that testosterone in humans acts as a fetal programming mechanism on the reward system and influences behavioral approach tendencies later in life. As a mechanism influencing atypical development, FT might be important across a range of neuropsychiatric conditions that asymmetrically affect the sexes....conduct disorder, psychopathy, attention-deficit/hyperactivity disorder, substance abuse, depression, bipolar disorder, cluster B personality disorders, intermittent explosive disorder, autism.
Research in rats suggests that doubling the blood testosterone level does not produce a measurable increase in the testosterone in the fetus. That same research also indicates that elevated testosterone in the mother hampers the placenta's ability to deliver amino acids (protein) to the developing fetus. Higher testosterone levels result in premature and low-birth weight newborns.
I wonder if any scientists will have the courage to follow the cognitive/emotional/physical development of the children born to women who are "transitioning".